Engineered extracellular vesicles from human placental mesenchymal stem cells attenuate abdominal aortic aneurysm formation by inhibiting Nat10-mediated vascular smooth muscle cell senescence
Quick Facts
- Publication title: Engineered extracellular vesicles from human placental mesenchymal stem cells attenuate abdominal aortic aneurysm formation by inhibiting Nat10-mediated vascular smooth muscle cell senescence
- Journal: Biomaterials
- Year: 2026
- DOI: 10.1016/j.biomaterials.2026.124109
- PMID/PMCID: 41791327
Research overview
Human placental mesenchymal stem cell-derived extracellular vesicles (hPMSC-EVs) have demonstrated anti-senescence effects across various diseases. However, their role in abdominal aortic aneurysm (AAA), a degenerative aortic pathology, remains unexplored.
Key findings
Human placental mesenchymal stem cell-derived extracellular vesicles (hPMSC-EVs) have demonstrated anti-senescence effects across various diseases.
However, their role in abdominal aortic aneurysm (AAA), a degenerative aortic pathology, remains unexplored.
Our findings demonstrated that hPMSC-EVs effectively delay vascular smooth muscle cell (VSMC) senescence.
In vivo, modification of hPMSC-EVs with an osteopontin(OPN)-targeted peptide facilitated precise homing to aneurysmal sites and improved local retention.
Echo Biotech Role
Echo Biotech contributed targeting-peptide/surface modification, custom synthesis or technical co-development; the study also used or cited ExoBrooch™.
Related platforms: Echosome®, Research Reagents & Tools
Related services and capabilities: Targeting Peptide / Surface Modification, Custom Ligand / Peptide-Lipid Synthesis, Research Reagent / Product Supply
Related products or reagents: ExoBrooch™ Targeting Peptide/Lipid Anchor Kit
References
Original publication: Engineered extracellular vesicles from human placental mesenchymal stem cells attenuate abdominal aortic aneurysm formation by inhibiting Nat10-mediated vascular smooth muscle cell senescence Biomaterials. 2026. DOI: 10.1016/j.biomaterials.2026.124109. PMID/PMCID: 41791327.