Tumor tissue derived extracellular vesicles promote diabetic wound healing

Quick Facts

  • Publication title: Tumor tissue derived extracellular vesicles promote diabetic wound healing
  • Journal: J DIABETES COMPLICAT
  • Year: 2023
  • DOI: 10.2147/IJN.S522994
  • PMID/PMCID: 40837696; PMC12363982

Research overview

This study aims to explore the effects of TNF-α-preconditioned ADSCs-exos (T-exos) on restoring DC function and accelerating wound healing in T2DM. By synergistically dampening inflammation and enhancing tissue repair, T-exos exhibit significant potential for clinical application in T2DM wounds.

Key findings

Under high glucose conditions, T-exos suppressed DC activation, as evidenced by reduced CD80/CD86 expression and NLRP3 inflammasome activity.

In vivo studies showed that T-exos promoted wound closure in T2DM mice, enhancing collagen deposition, angiogenesis, and fibroblast proliferation.

Mechanistically, TNF-α enriched miR-146a-5p in exosomes, which targeted TXNIP to inhibit NLRP3 inflammasome activation in DCs.

Knockdown of miR-146a-5p abolished these effects in vitro and the therapeutic effect of T-exos on wound healing in vivo.

Study design

T-exos were isolated from ADSCs pretreated with TNF-α.

The regulatory effects of T-exos on DC immune responses under high glucose conditions were assessed.

Subsequently, the roles of DCs treated with T-exos in diabetic wound healing were evaluated.

Echo Biotech Role

Echo Biotech contributed EV isolation and purification, EV characterization; the study also used or cited Exosupur®.

Related platforms: Exoomics®, Research Reagents & Tools

Related services and capabilities: Tissue EV Isolation & Purification, EV Basic Characterization (TEM/NTA/WB), Research Reagent / Product Supply

Related products or reagents: Exosupur® EV Isolation/Purification Kit

References

Original publication: Tumor tissue derived extracellular vesicles promote diabetic wound healing Journal of diabetes and its complications. 2023. DOI: 10.2147/IJN.S522994. PMID/PMCID: 40837696; PMC12363982.